Can you explain chronic kidney disease (CKD), including its definition, staging, causes, risk factors, evaluation, and management?

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Chronic Kidney Disease (CKD): Comprehensive Overview

Definition and Diagnostic Criteria

CKD is diagnosed when kidney abnormalities persist for more than 3 months, defined by either an estimated glomerular filtration rate (eGFR) <60 mL/min/1.73 m² or evidence of kidney damage (such as albuminuria ≥30 mg/g, abnormal imaging, or biopsy findings) at any eGFR level. 1, 2

The 3-month persistence requirement is critical—it distinguishes chronic from acute kidney injury and requires documentation on at least two separate occasions. 1, 2

Key Diagnostic Tests

  • Serum creatinine to calculate eGFR using the CKD-EPI equation (preferred over MDRD due to less bias, especially at eGFR ≥60 mL/min/1.73 m²) 1, 3, 2
  • Urine albumin-to-creatinine ratio (UACR) on a random spot urine sample 1
  • Urinalysis with microscopy to detect hematuria, cellular casts, or other abnormalities 1

When creatinine-based eGFR is uncertain (e.g., extremes of muscle mass), confirm with cystatin C-based or combined creatinine-cystatin C equations. 1, 3

Staging System: The CGA Classification

CKD must be classified using the complete CGA system: Cause, GFR category, and Albuminuria category—never stage by GFR alone. 3, 2

GFR Categories (G Stages)

Stage eGFR (mL/min/1.73 m²) Description
G1 ≥90 Normal/high function (requires evidence of kidney damage for CKD diagnosis)
G2 60-89 Mildly decreased (requires evidence of kidney damage for CKD diagnosis)
G3a 45-59 Mild-to-moderate decrease
G3b 30-44 Moderate-to-severe decrease
G4 15-29 Severe decrease
G5 <15 Kidney failure

4, 1, 3

Critical point: For G1 and G2, you cannot diagnose CKD based on eGFR alone—you must document kidney damage (albuminuria, imaging abnormality, biopsy findings, or abnormal sediment). 3, 2

The subdivision of Stage 3 into 3a and 3b is mandatory because these ranges have markedly different mortality, cardiovascular risk, and progression profiles. 3, 2

Albuminuria Categories (A Stages)

Category UACR (mg/g) Description
A1 <30 Normal to mildly increased
A2 30-300 Moderately increased
A3 >300 Severely increased (includes nephrotic range)

1, 3

The 30 mg/g threshold represents approximately 3 times the normal value and independently predicts CKD complications, cardiovascular mortality, and progression to kidney failure. 3, 2

Combined Risk Stratification (KDIGO Heat Map)

The combination of GFR and albuminuria categories determines overall risk:

  • Low risk (green): G1-G2 with A1 3
  • Moderately increased risk (yellow): G1-G2 with A2, or G3a with A1 3
  • High risk (orange): G1-G2 with A3, or G3a with A2, or G3b with A1 3
  • Very high risk (red): G3a with A3, or G3b with A2-A3, or any G4-G5 (regardless of albuminuria) 3

This risk stratification directly guides monitoring frequency, blood pressure targets, nephrology referral timing, and cardiovascular risk-reduction strategies. 3

Causes and Risk Factors

Primary Causes

In developed countries, the two dominant causes are:

  • Diabetes mellitus (diabetic kidney disease develops in 20-40% of diabetic patients) 4, 5, 6
  • Hypertension (hypertensive nephrosclerosis) 5, 6

Other important causes include glomerulonephritis, polycystic kidney disease, and chronic interstitial nephritis. 3

Risk Factors for Development

  • Age >60 years 7, 8
  • Diabetes mellitus 5, 8
  • Hypertension 5, 8
  • Cardiovascular disease 7, 8
  • Family history of kidney disease 1, 8
  • Exposure to nephrotoxic medications (NSAIDs, certain antibiotics, contrast agents) 5, 8

Risk Factors for Progression

  • Elevated blood pressure 1
  • Hyperglycemia 4
  • Higher degrees of albuminuria 4, 5
  • Rapid eGFR decline (≥5 mL/min/1.73 m²/year or ≥30% decrease over 2 years) 2, 5

Clinical Evaluation

Initial Assessment

When CKD is suspected or detected, perform:

  • Focused history: duration of diabetes or hypertension, family history of kidney disease, medication review (especially NSAIDs, ACE inhibitors, ARBs), symptoms of uremia (nausea, pruritus, altered mental status), volume status 1, 5
  • Physical examination: blood pressure measurement, volume assessment (edema, jugular venous pressure), signs of uremia 1
  • Laboratory tests:
    • Complete blood count (to assess anemia) 1
    • Comprehensive metabolic panel (electrolytes, calcium, phosphate, bicarbonate) 1
    • Lipid panel 4
    • Urinalysis with microscopy 1
    • UACR 1
  • Imaging: Renal ultrasound to assess kidney size, echogenicity, and rule out obstruction 1

Monitoring Frequency by Stage

  • G1-G2 (with kidney damage): Annual monitoring 3
  • G3a: Every 6-12 months depending on albuminuria level 1, 3
  • G3b: Every 3-6 months 3
  • G4-G5: Every 1-3 months 3

Management by Stage

Stage 1-2 (eGFR ≥60 with kidney damage)

Primary goals: Slow progression, reduce cardiovascular risk, treat underlying cause

  • Blood pressure control: Target <130/80 mmHg, especially with albuminuria ≥300 mg/g 1, 5
  • ACE inhibitors or ARBs: First-line for patients with UACR ≥30 mg/g 1
  • Glycemic control: HbA1c target individualized but generally <7% in diabetics 4
  • Cardiovascular risk reduction: Statin therapy for lipid management 4, 5
  • Lifestyle modifications: Sodium restriction, smoking cessation, weight management 5
  • Avoid nephrotoxins: NSAIDs, aminoglycosides, contrast agents when possible 5, 8

Stage 3a (eGFR 45-59)

Continue all Stage 1-2 measures, plus:

  • Intensify blood pressure and proteinuria control 1
  • Begin monitoring for complications: Anemia, mineral bone disease 4, 1
  • Adjust medication doses according to eGFR 5, 8
  • Consider nephrology referral if UACR ≥300 mg/g or rapid progression 1, 5

Stage 3b (eGFR 30-44)

Continue all previous measures, plus:

  • Systematic evaluation for CKD complications:
    • Anemia (hemoglobin monitoring, consider erythropoiesis-stimulating agents if indicated) 4, 6
    • Mineral bone disease (calcium, phosphate, PTH, vitamin D levels) 4, 5
    • Metabolic acidosis (serum bicarbonate) 5
    • Hyperkalemia 5
  • Strict medication dose adjustments 5, 8
  • Nephrology referral strongly recommended 1, 5

Stage 4 (eGFR 15-29)

All Stage 4 patients require nephrology referral. 1, 7, 5

  • Intensive management of complications 4, 1
  • Preparation for renal replacement therapy:
    • Education about dialysis modalities (hemodialysis, peritoneal dialysis) and transplantation 7
    • Vascular access planning (arteriovenous fistula creation 6-12 months before anticipated dialysis need) 7
    • Transplant evaluation if appropriate 7
  • Dietary modifications: Protein restriction (0.6-0.8 g/kg/day), potassium and phosphate restriction 7

Stage 5 (eGFR <15)

Kidney failure requiring renal replacement therapy when uremic symptoms develop or metabolic complications become unmanageable. 4, 3

  • Initiate dialysis when indicated (uremic symptoms, refractory hyperkalemia, metabolic acidosis, volume overload, pericarditis) 4, 3
  • Continue transplant evaluation if candidate 7

Specific Therapies to Slow Progression

Renin-Angiotensin System Blockade

ACE inhibitors or ARBs are first-line for patients with UACR ≥300 mg/g and/or eGFR <60 mL/min/1.73 m². 1

These agents reduce proteinuria and slow CKD progression independent of blood pressure effects. 5 Do not combine ACE inhibitors with ARBs—this increases adverse events without additional benefit. 5

SGLT2 Inhibitors

SGLT2 inhibitors reduce CKD progression and cardiovascular events in patients with diabetes and CKD. 1

These agents provide kidney protection beyond glycemic control and should be considered in diabetic CKD patients with eGFR ≥20 mL/min/1.73 m². 4, 1

Blood Pressure Management

Target blood pressure <130/80 mmHg, particularly in patients with albuminuria ≥300 mg/g. 1, 5

Lower targets may be appropriate for patients with higher degrees of proteinuria, but monitor for acute eGFR decline when initiating or intensifying therapy. 4

Nephrology Referral Criteria

Refer to nephrology when:

  • eGFR <30 mL/min/1.73 m² (all Stage 4-5 patients) 1, 7, 5
  • UACR ≥300 mg/g 1, 5
  • Rapid eGFR decline (≥30% decrease over 2 years or ≥5 mL/min/1.73 m²/year) 2, 5
  • Difficulty managing CKD complications (anemia, mineral bone disease, metabolic acidosis) 1, 5
  • Uncertain etiology requiring kidney biopsy 5
  • Active urinary sediment (dysmorphic RBCs, RBC casts) suggesting glomerulonephritis 5

Early referral (at Stage 3b-4) improves long-term outcomes and reduces costs by allowing adequate preparation for renal replacement therapy. 7

Common Pitfalls and Caveats

Diagnostic Errors

  • Never diagnose CKD on a single abnormal test—the abnormality must be documented on at least two occasions ≥3 months apart. 3, 2
  • Do not rely on serum creatinine alone—a "normal" creatinine (e.g., 1.2 mg/dL) in an elderly patient with low muscle mass may represent significant kidney dysfunction (eGFR 40-50 mL/min/1.73 m²). 2
  • Always classify using the complete CGA system—stating "Stage 3 CKD" without specifying 3a vs 3b and albuminuria category provides incomplete risk stratification. 3, 2

Management Errors

  • Avoid therapeutic nihilism—patients with CKD derive equal or greater benefit from evidence-based cardiovascular therapies (statins, blood pressure control) compared to the general population. 9
  • Do not withhold ACE inhibitors/ARBs due to mild creatinine elevation—a 20-30% increase in creatinine after initiation is acceptable and does not indicate harm. 5 However, increases >30% warrant investigation for renal artery stenosis or volume depletion. 5
  • Adjust medication doses according to eGFR—many antibiotics, oral hypoglycemics, and other drugs require dose reduction to prevent toxicity. 5, 8
  • Screen for and avoid nephrotoxins—NSAIDs are particularly harmful in CKD and should be avoided. 5, 8

Acute Kidney Injury Superimposed on CKD

Patients with CKD are at higher risk for acute kidney injury from volume depletion, contrast agents, or medications. 4 When eGFR drops acutely, repeat measurement within days (not months) to distinguish acute from chronic decline. 4

Epidemiology and Public Health Impact

CKD affects 8-16% of the global population and is the 16th leading cause of years of life lost worldwide. 5, 6 However, less than 5% of patients with early CKD are aware of their disease. 5

Patients with CKD are 5-10 times more likely to die (primarily from cardiovascular disease) than to progress to end-stage kidney disease. 6, 9 This cardiovascular risk increases exponentially as kidney function declines and persists even after adjustment for traditional risk factors. 9

CKD should be considered a coronary heart disease risk equivalent, warranting aggressive cardiovascular risk factor management. 4, 9

References

Guideline

Chronic Kidney Disease Diagnosis and Treatment

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Chronic Kidney Disease Diagnosis and Staging

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Chronic Kidney Disease Staging and Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Chronic Kidney Disease.

Lancet (London, England), 2017

Research

[Advanced chronic kidney disease].

Nefrologia : publicacion oficial de la Sociedad Espanola Nefrologia, 2008

Research

Kidney Disease: Chronic Kidney Disease.

FP essentials, 2021

Research

Cardiovascular risk in chronic kidney disease.

Kidney international. Supplement, 2004

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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