What are the benefits, standard dosing, and adverse effects of prednisone or dexamethasone in induction therapy for acute lymphoblastic leukemia, and how should they be monitored and managed?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board

Educational purpose only • Exercise caution as content is pending human review

Article Review Status

Submitted

Under Review

Approved

Last updated: February 28, 2026 • View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Related Questions

What is the comparative teratogenicity of prednisolone (corticosteroid) and dexamethasone (corticosteroid) during pregnancy?

Can dexamethasone replace prednisolone to achieve better central nervous system penetration in acute lymphoblastic leukaemia patients?

How is prednisone used (dose, schedule, duration, side‑effect profile) in induction therapy for acute lymphoblastic leukemia, and when should dexamethasone be used instead?

Is it acceptable to substitute prednisolone with prednisone in patients (pts) with Acute Myeloid Leukemia (AML)?

Is it safe for someone with inflammation to take prednisone and use butane?

What does a down‑trending anti‑factor Xa level indicate after it was previously elevated?

At 22 years old, 56 kg, 173 cm (BMI ≈ 18.7), with negative stool parasite and protozoa examinations, negative Helicobacter pylori stool antigen, normal fecal calprotectin, and normal pancreatic elastase, could I still have an organic gastrointestinal disease or is this most consistent with a functional gastrointestinal disorder?

Why do we trend anti‑Factor Xa activity in patients receiving heparin?

How is prednisone used (dose, schedule, duration, side‑effect profile) in induction therapy for acute lymphoblastic leukemia, and when should dexamethasone be used instead?

Is it safe or advisable to combine venlafaxine 75 mg daily with escitalopram 10 mg daily?

I am a 22‑year‑old, 56 kg, 173 cm individual with normal fecal calprotectin, normal pancreatic elastase, negative stool parasites, negative Helicobacter pylori antigen, and I experience post‑prandial flushing, heat sensations, temperature‑dependent symptoms, and hormonal fluctuations; could this be due to mast‑cell activation/histamine release, autonomic (vagal) dysfunction, visceral hypersensitivity, or stress/CRH‑mediated gut activation?

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.