What are the criteria for Hyperviscosity Syndrome?

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From the Guidelines

Hyperviscosity syndrome is diagnosed based on a combination of clinical symptoms and laboratory findings, including serum viscosity greater than 4.0 centipoise, visual disturbances, neurological symptoms, and bleeding, with treatment initiated for symptomatic patients, particularly those with hyperviscosity, neuropathy, or other indicative symptoms 1. The criteria for hyperviscosity syndrome include:

  • Serum viscosity greater than 4.0 centipoise (normal is 1.4-1.8)
  • Clinical manifestations such as:
    • Visual disturbances (blurred vision, retinal hemorrhages)
    • Neurological symptoms (headaches, dizziness, confusion)
    • Bleeding (epistaxis, gingival bleeding)
  • Laboratory findings showing elevated protein levels, particularly in conditions like:
    • Waldenström macroglobulinemia (IgM > 3 g/dL)
    • Multiple myeloma (IgA or IgG > 4 g/dL)
    • Polycythemia (hematocrit > 60%) According to the NCCN guidelines, treatment should be initiated for patients with a diagnosis of WM/LPL only in those who are symptomatic, with indicative symptoms including hyperviscosity, neuropathy, symptomatic adenopathy or organomegaly, amyloidosis, cryoglobulinemia, cold agglutinin disease, anemia, B symptoms, and presence of cytopenia 1. Management typically involves plasmapheresis for immediate relief of symptoms, followed by treating the underlying condition causing the hyperviscosity, such as chemotherapy for plasma cell dyscrasias or phlebotomy for polycythemia 1. Key considerations in the management of hyperviscosity syndrome include:
  • Initiating treatment for symptomatic patients, particularly those with hyperviscosity or other indicative symptoms
  • Using plasmapheresis for immediate relief of symptoms
  • Treating the underlying condition causing the hyperviscosity
  • Avoiding agents that limit future treatment options, such as continuous oral alkylator therapy or nucleoside analogues, particularly in younger patients who may be potential SCT candidates 1.

From the Research

Hyperviscosity Syndrome Criteria

The criteria for hyperviscosity syndrome include:

  • Clinical manifestations such as neurological impairment, visual disturbance, and bleeding 2
  • Measurement of plasma or serum viscosity by a viscometer to assess diagnosis 2
  • Funduscopic examination to identify abnormalities correlated with abnormal plasma viscosity 2
  • Etiologies including various conditions, with symptomatic hyperviscosity more common in Waldenström's macroglobulinemia and multiple myeloma 2

Diagnosis and Treatment

Diagnosis and treatment of hyperviscosity syndrome involve:

  • Viscosity measurements to confirm diagnosis, although therapy can be initiated without these measurements if clinical presentation is typical 3
  • Plasma exchange as the therapy of choice for patients with hemorrhagic or central nervous system manifestations 3
  • Phlebotomy for patients with extreme elevation of red cell and white cell count 3
  • Therapeutic apheresis, including plasmapheresis and cytapheresis, to remove abnormal plasma components or excess blood cells 4

Associated Conditions

Hyperviscosity syndrome is associated with various conditions, including:

  • Monoclonal and polyclonal disorders with elevation of immunoglobulins 3
  • Waldenström's macroglobulinemia and multiple myeloma 2, 4
  • Polycythemia vera, essential thrombocytosis, and sickle cell disease 4, 5
  • Leukemias with hyperleukocytosis 4

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

[Hyperviscosity syndrome].

La Revue de medecine interne, 2015

Research

Correction of hyperviscosity by apheresis.

Seminars in thrombosis and hemostasis, 2003

Research

Hyperviscosity in polycythemia vera and other red cell abnormalities.

Seminars in thrombosis and hemostasis, 2003

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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