What diuretic does not induce gout?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: June 10, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

From the FDA Drug Label

WARNINGS Hyperkalemia Like other potassium-conserving agents, amiloride may cause hyperkalemia (serum potassium levels greater than 5. 5 mEq per liter) which, if uncorrected, is potentially fatal.

Diabetes Mellitus In diabetic patients, hyperkalemia has been reported with the use of all potassium-conserving diuretics, including amiloride HCl, even in patients without evidence of diabetic nephropathy.

The diuretic that does not induce gout is not directly mentioned in the provided drug label. However, amiloride is a potassium-conserving diuretic, and its use is associated with hyperkalemia rather than hypokalemia, which can contribute to gout.

  • Amiloride may be considered as it does not cause hypokalemia, a common side effect of other diuretics that can induce gout 1.

From the Research

Potassium-sparing diuretics, such as spironolactone, eplerenone, amiloride, and triamterene, do not typically induce gout and are considered safer options for patients with a history of gout or hyperuricemia. These medications work by blocking sodium reabsorption in the distal tubule without affecting uric acid handling in the kidneys, unlike thiazide and loop diuretics which increase uric acid reabsorption in the proximal tubule leading to hyperuricemia 2, 3.

The most recent and highest quality study, a randomized clinical trial published in 2025, compared the efficacy of spironolactone and amiloride in patients with resistant hypertension, and found that amiloride was noninferior to spironolactone in lowering home-measured systolic blood pressure, with no cases of gynecomastia in either group 4.

Key characteristics of potassium-sparing diuretics include:

  • Blocking sodium reabsorption in the distal tubule
  • Not affecting uric acid handling in the kidneys
  • Being safer options for patients with a history of gout or hyperuricemia
  • Possibly being combined with a uric acid lowering therapy if needed for blood pressure management

If a patient requires diuretic therapy but has gout, a potassium-sparing diuretic would be the preferred choice. For patients who must use thiazide or loop diuretics despite gout risk, prophylactic allopurinol may be considered to prevent gout attacks, as supported by studies on gout management 5, 6.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.