How is Multiple System Atrophy (MSA) diagnosed?

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Last updated: July 25, 2025View editorial policy

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Diagnosis of Multiple System Atrophy (MSA)

Multiple System Atrophy (MSA) is diagnosed through a combination of clinical evaluation of characteristic symptoms, neuroimaging findings, and autonomic function testing, with definitive diagnosis requiring neuropathological confirmation of alpha-synuclein-positive glial cytoplasmic inclusions at autopsy.

Clinical Diagnostic Criteria

The diagnosis of MSA relies on established consensus criteria that categorize patients into three levels of diagnostic certainty:

Definite MSA

  • Requires neuropathological demonstration of CNS alpha-synuclein-positive glial cytoplasmic inclusions with neurodegenerative changes in striatonigral or olivopontocerebellar structures 1

Probable MSA

  • Sporadic, progressive adult-onset disorder including:
    • Rigorously defined autonomic failure (orthostatic hypotension)
    • PLUS either:
      • Poorly levodopa-responsive parkinsonism (MSA-P subtype)
      • OR cerebellar ataxia (MSA-C subtype) 1

Possible MSA

  • Sporadic, progressive adult-onset disease including:
    • Parkinsonism or cerebellar ataxia
    • PLUS at least one feature suggesting autonomic dysfunction (e.g., urinary incontinence)
    • PLUS one other supporting feature (clinical or neuroimaging) 1

Key Clinical Features to Evaluate

Motor Symptoms

  • Parkinsonism: Bradykinesia, rigidity, postural instability, and resting tremor (less common than in Parkinson's disease)
  • Cerebellar signs: Gait ataxia, limb ataxia, dysarthria, nystagmus
  • Pyramidal signs: Hyperreflexia, extensor plantar responses

Autonomic Dysfunction

  • Orthostatic hypotension (≥30 mmHg systolic or ≥15 mmHg diastolic drop within 3 minutes of standing)
  • Urinary symptoms: Incontinence, retention, urgency
  • Sexual dysfunction: Erectile dysfunction in males
  • Constipation
  • Anhidrosis or hyperhidrosis

Red Flags for MSA Diagnosis

  • Poor or transient response to levodopa therapy
  • Early falls and postural instability
  • Rapid disease progression
  • Early and severe autonomic failure
  • Stridor (particularly during sleep)

Diagnostic Investigations

Neuroimaging

  • MRI Brain: The optimal imaging modality for MSA diagnosis 2
    • MSA-P: Putaminal atrophy, hypointensity on T2-weighted images, "hot cross bun" sign
    • MSA-C: Cerebellar and pontine atrophy, middle cerebellar peduncle hyperintensity
    • 3T MRI is preferable over 1.5T for better resolution 2

Autonomic Function Testing

  • Cardiovascular autonomic testing to document orthostatic hypotension
  • Urodynamic studies to evaluate bladder dysfunction
  • Sudomotor function tests

Nuclear Medicine Imaging

  • Ioflupane SPECT/CT (DaTscan): Demonstrates decreased radiotracer uptake in the striatum, but cannot differentiate MSA from other parkinsonian syndromes 2
  • Cardiac sympathetic imaging: MIBG scintigraphy may help differentiate MSA from Parkinson's disease

Other Supporting Tests

  • Polysomnography to detect REM sleep behavior disorder and sleep-disordered breathing
  • Neurophysiological studies (e.g., sphincter EMG)

Differential Diagnosis

The main differential diagnoses to consider include:

  • Parkinson's disease
  • Progressive supranuclear palsy (PSP)
  • Corticobasal degeneration (CBD)
  • Pure autonomic failure
  • Spinocerebellar ataxias
  • Vascular parkinsonism

Diagnostic Challenges and Pitfalls

  1. Early diagnosis is difficult: Patients may initially present with isolated autonomic failure or motor symptoms, making early diagnosis challenging 3

  2. Misdiagnosis is common: Up to 20-30% of patients diagnosed with MSA in life have a different pathology at autopsy

  3. Atypical features: Some patients may present with features not typically associated with MSA, such as dementia, hallucinations, or vertical gaze palsy 3

  4. Variable levodopa response: While often described as "poorly levodopa-responsive," approximately 40-60% of MSA patients with predominant parkinsonian features may show some response to levodopa, especially early in the disease course 4

Emerging Diagnostic Approaches

  • Advanced MRI techniques: Diffusion tensor imaging, voxel-based morphometry
  • Fluid biomarkers: Alpha-synuclein in cerebrospinal fluid
  • Skin biopsy: Detection of phosphorylated alpha-synuclein deposits
  • Retinal imaging biomarkers

By systematically evaluating clinical features and using appropriate diagnostic tests, clinicians can improve the accuracy of MSA diagnosis, which is crucial for proper management, counseling, and potential enrollment in clinical trials.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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