What are the side effects of Indapamide?

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Side Effects of Indapamide

Indapamide's most common side effects include electrolyte disturbances (particularly hypokalemia), metabolic changes, and mild gastrointestinal symptoms. 1

Common Side Effects

  • Electrolyte disturbances: Hypokalemia is the most significant concern, with studies showing 47% of patients on indapamide 2.5 mg having at least one potassium value below 3.5 mEq/L 1
  • Metabolic effects: Hyperuricemia (average increase of 0.69-1.0 mg/dL), glucose intolerance, and acid-base disturbances 1, 2
  • Gastrointestinal symptoms: Nausea, constipation, gastric irritation, and abdominal pain 1
  • Central nervous system: Headache, dizziness, and nervousness 1
  • Other common effects: Fatigue, asthenia, and peripheral edema 1

Electrolyte Abnormalities

  • Potassium: Hypokalemia risk increases with higher doses, brisk diuresis, and concomitant use of corticosteroids 1
  • Sodium: Hyponatremia may occur, particularly in edematous patients 1
  • Magnesium: Increased urinary excretion may lead to hypomagnesemia 1
  • Calcium: Unlike other diuretics, indapamide causes only slight increases in serum calcium after prolonged treatment 1

Cardiovascular Effects

  • Blood pressure: Orthostatic hypotension may occur, particularly in elderly patients or with volume depletion 2
  • Cardiac effects: Indapamide has fewer negative effects on cardiac function compared to some other diuretics 3

Metabolic and Endocrine Effects

  • Uric acid: Serum concentrations typically increase by 0.69-1.0 mg/dL, potentially precipitating gout in susceptible individuals 1
  • Glucose metabolism: May cause glucose intolerance and alter insulin requirements in diabetic patients 1
  • Lipid profile: Unlike some diuretics, indapamide has minimal adverse effects on lipid metabolism 3

Renal Effects

  • Renal function: Should be used with caution in severe renal disease as reduced plasma volume may exacerbate azotemia 1
  • Urinary calcium: Indapamide reduces urinary calcium excretion by 20-50% depending on the patient population 4

Hepatic Effects

  • Liver function: Should be used cautiously in patients with impaired hepatic function as minor fluid/electrolyte imbalances may precipitate hepatic coma 1
  • Rare reactions: Jaundice (intrahepatic cholestatic), hepatitis, and abnormal liver function tests have been reported 1

Dermatologic and Hypersensitivity Reactions

  • Skin reactions: Rash, hives, pruritus, and photosensitivity 1
  • Severe reactions: Erythema multiforme, Stevens-Johnson Syndrome, and vasculitis have been reported but are rare 1

Hematologic Effects

  • Blood disorders: Rare reports of agranulocytosis, leukopenia, thrombocytopenia, and aplastic anemia 1

Dosage Considerations

  • Initial dosing: The recommended initial dose is 2.5 mg once daily, taken in the morning 2
  • Maximum dose: The maximum recommended daily dose is 2.5 mg 2
  • Monitoring: Blood pressure, renal function, and electrolytes should be checked 1-2 weeks after each dose increment, at 3 months, and subsequently at 6-month intervals 2

Special Populations

  • Renal impairment: Monitoring of renal function is essential; withhold or discontinue if progressive renal impairment is observed 1
  • Hepatic impairment: Use with caution due to risk of precipitating hepatic coma 1
  • Elderly: May be more sensitive to the hypotensive and electrolyte effects 2

Contraindications and Precautions

  • Severe renal disease: Use with caution due to risk of azotemia 1
  • Impaired hepatic function: Use with caution due to risk of precipitating hepatic coma 1
  • Systemic lupus erythematosus: May exacerbate or activate the condition 1
  • Acute angle-closure glaucoma: Sulfonamide-derivative drugs like indapamide can cause idiosyncratic reactions resulting in acute angle-closure glaucoma 1

Drug Interactions

  • Other antihypertensives: May potentiate the action of other antihypertensive drugs 1
  • Lithium: May increase risk of lithium toxicity 1
  • NSAIDs: Should be avoided as they may reduce the antihypertensive effect and increase risk of renal impairment 2
  • Potassium-sparing diuretics: Should be avoided during initiation of therapy 2

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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