Can Enfortumab (enfortumab vedotin) cause hyperglycemia?

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Last updated: October 27, 2025View editorial policy

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Enfortumab Vedotin and Hyperglycemia

Yes, enfortumab vedotin can cause hyperglycemia, which can be severe and potentially lead to diabetic ketoacidosis in some patients. 1

Evidence for Hyperglycemia with Enfortumab Vedotin

  • Hyperglycemia is explicitly listed as a significant adverse effect in the FDA label for PADCEV (enfortumab vedotin), with a specific warning that diabetic ketoacidosis may occur in patients with and without preexisting diabetes mellitus, which may be fatal 1
  • In the EV-302/KEYNOTE-39A trial, grade 3 hyperglycemia occurred in 6.1% of patients treated with enfortumab vedotin plus pembrolizumab 2
  • The European Association of Urology guidelines (2025) specifically list hyperglycemia as one of the key toxicities of enfortumab vedotin, alongside rash, neuropathy, and ocular disorders 2
  • In the EV-201 Cohort 2 trial, hyperglycemia was reported in 16% of patients, with grade 3-4 hyperglycemia occurring in 9% of patients 1
  • Laboratory abnormalities from clinical trials show increased glucose (non-fasting) in 33-36% of patients, with grade 3-4 elevations in 9-13% of patients 1

Mechanism and Risk Factors

  • While the exact mechanism is not fully elucidated in the evidence provided, case reports suggest that enfortumab vedotin may cause insulin resistance 3, 4
  • Patients with pre-existing diabetes may be at higher risk, as demonstrated in case reports where patients with diabetes experienced severe hyperglycemia after enfortumab vedotin administration 4
  • However, hyperglycemia and even diabetic ketoacidosis can occur in patients without pre-existing diabetes 5

Severity and Clinical Implications

  • Hyperglycemia with enfortumab vedotin can range from mild elevations to severe, potentially fatal diabetic ketoacidosis 1, 5
  • Case reports describe instances of severe insulin resistance, diabetic ketoacidosis, and acute kidney injury associated with enfortumab vedotin treatment 3, 4, 5
  • One case report described a patient with no prior history of diabetes who developed fatal diabetic ketoacidosis after the second dose of enfortumab vedotin, despite aggressive treatment 5

Management Recommendations

  • The FDA label recommends closely monitoring blood glucose levels in patients with, or at risk for, diabetes mellitus or hyperglycemia 1
  • Withhold enfortumab vedotin if blood glucose is >250 mg/dL 1
  • Patients with uncontrolled diabetes may be ineligible for enfortumab vedotin treatment, according to the European Association of Urology guidelines 2
  • For patients who develop hyperglycemia while on enfortumab vedotin, management should follow standard protocols for hyperglycemia, with metformin typically used as first-line therapy for mild cases 6, 7
  • For severe hyperglycemia (blood glucose ≥250 mg/dL), insulin therapy may be required 7

Clinical Pearls and Pitfalls

  • Pitfall to avoid: Failing to screen for and monitor blood glucose in patients receiving enfortumab vedotin, especially those with risk factors for diabetes 8
  • Pitfall to avoid: Underestimating the potential severity of hyperglycemia, which can progress rapidly to diabetic ketoacidosis even in patients without pre-existing diabetes 5
  • Clinical pearl: Consider baseline diabetes screening before initiating enfortumab vedotin therapy 5
  • Clinical pearl: Educate patients about symptoms of hyperglycemia and the importance of reporting them promptly 8
  • Clinical pearl: Have a lower threshold for hospital admission for patients on enfortumab vedotin who develop significant hyperglycemia, as it may progress rapidly 5

Despite the risk of hyperglycemia, enfortumab vedotin (particularly in combination with pembrolizumab) remains a preferred treatment option for eligible patients with advanced urothelial cancer due to its significant survival benefits 2.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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