Does glutamine supplementation decrease calprotectin levels in patients with inflammatory bowel disease?

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Last updated: November 15, 2025View editorial policy

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Glutamine Does Not Decrease Calprotectin in Inflammatory Bowel Disease

Based on the best available evidence, glutamine supplementation does not reduce calprotectin levels or improve inflammatory markers in patients with inflammatory bowel disease, regardless of the route of administration.

Evidence from Systematic Reviews

The most comprehensive and recent evidence comes from a 2021 systematic review that specifically evaluated glutamine supplementation in IBD patients 1. This review found:

  • No effect on inflammatory markers: Glutamine supplementation showed no impact on inflammation markers, including calprotectin, in IBD patients 1
  • No effect on disease activity: The review found no changes in disease course, intestinal permeability, morphology, or disease activity scores 1
  • Route of administration irrelevant: These null findings were consistent across oral (21-30g or 0.5g/kg), enteral (7.87-8.3g/100g), and parenteral (0.3g/kg) routes 1

Guideline Recommendations

ESPEN guidelines explicitly state there is insufficient evidence to recommend glutamine supplementation in IBD:

  • For Crohn's disease, only two small randomized trials have compared glutamine-enriched formulas to standard formulas or placebo, and neither demonstrated any clinical benefit 2
  • Despite theoretical benefits from animal models showing reduced intestinal damage, human trials have failed to translate these findings into meaningful clinical outcomes 2
  • The 2009 ESPEN gastroenterology guidelines concluded there is currently insufficient evidence to recommend glutamine use in CD patients 2

Additional Supporting Evidence

A 2016 Cochrane review reinforced these findings 3:

  • Two small RCTs (42 total patients) found no statistically significant differences in clinical remission rates with glutamine supplementation 3
  • No changes in intestinal permeability were observed in either study 3
  • The review concluded there is insufficient evidence to support glutamine for induction of remission in Crohn's disease 3

Understanding Calprotectin Context

While calprotectin is a validated marker of intestinal inflammation in IBD 4, 5:

  • Calprotectin levels can distinguish active inflammation from IBS-like symptoms in IBD patients in remission 4
  • Higher calprotectin values are characteristic of inflammatory bowel disease compared to functional disorders 5
  • However, glutamine has not been shown to reduce this inflammatory marker in clinical trials 1

Clinical Bottom Line

Do not use glutamine supplementation with the expectation of reducing calprotectin levels or controlling inflammation in IBD patients. The evidence consistently shows no benefit across multiple routes of administration and patient populations 1, 3. The theoretical rationale from animal studies has not translated into human clinical benefit 2, 6.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Glutamine for induction of remission in Crohn's disease.

The Cochrane database of systematic reviews, 2016

Research

Diagnostic value of calprotectin in irritable bowel syndrome and in inflammatory bowel disease.

Romanian journal of internal medicine = Revue roumaine de medecine interne, 2012

Guideline

Glutamine Supplementation and Intestinal Secretory IgA

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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