What causes appendicitis?

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What Causes Appendicitis

Appendicitis develops primarily from obstruction of the appendiceal lumen by fecaliths (hardened stool) or parasites, leading to increased intraluminal pressure, mucosal ischemia, and subsequent bacterial invasion. 1

Primary Pathophysiological Mechanism

The fundamental cause follows a well-established sequence:

  • Luminal obstruction is the initiating event, most commonly caused by fecaliths (hardened fecal matter) or parasites that block the narrow appendiceal opening 1
  • This obstruction creates a closed-loop system where continued mucus secretion increases intraluminal pressure 1
  • Rising pressure compromises venous and lymphatic drainage, causing mucosal ischemia and edema 1
  • Ischemic mucosa becomes vulnerable to bacterial invasion from normal intestinal flora, leading to inflammation and potential perforation 1

Contributing Factors and Disease Progression

The natural history follows three distinct stages:

  • Stage 1: Normal appendix 1
  • Stage 2: Uncomplicated acute appendicitis (approximately two-thirds of cases) 1
  • Stage 3: Complicated appendicitis with gangrene or perforation (perforation rates 16-40%) 1

Structural and Age-Related Factors

  • Appendicoliths (calcified fecal concretions) are strongly associated with progression to complicated disease and perforation 1
  • Elderly patients develop vascular sclerosis of the appendix with muscular layers infiltrated by fat, creating structural weakness and tendency toward early perforation 1
  • Lymphoid hyperplasia may contribute to obstruction, particularly in younger patients, though this represents only a small percentage of cases that progress to complete occlusion 2

Clinical Presentation Pattern

The characteristic pain migration reflects the underlying pathophysiology:

  • Pain typically begins periumbilically due to visceral innervation of the inflamed appendix 1
  • Pain then migrates to the right lower quadrant as the parietal peritoneum becomes inflamed 1
  • Additional findings that increase likelihood include fever, positive psoas sign, and migratory pain to the right lower quadrant 3

Risk Factors for Complicated Disease

Several factors significantly increase perforation risk:

  • Advanced age (>65 years) dramatically increases perforation rates to 18-70% compared to 3-29% in younger patients 1
  • Delayed presentation with median duration of approximately 4 days from symptom onset to care in complicated cases 1
  • Mortality risk increases threefold with each decade of age beyond 65 years 1

Geographic and Epidemiologic Considerations

  • Appendicitis accounts for 34.2% of all intra-abdominal infections worldwide, making it the most common cause of intra-abdominal sepsis 1
  • In resource-limited settings, parasitic infections (Entamoeba, Ascaris, Trichuris trichiura, Enterobius vermicularis, Salmonella Typhi) can mimic or cause appendicitis 3
  • Lifetime risk varies geographically: 9% in USA, 8% in Europe, 2% in Africa 1

Common Pitfalls

  • Atypical presentations are common, particularly in elderly patients and children, often resulting in delayed diagnosis 3
  • The presence of vomiting before pain onset makes appendicitis unlikely 3
  • Not all appendicitis progresses to perforation; some cases of simple appendicitis may be self-limiting, though this remains controversial 4

References

Guideline

Stages of Appendicitis and Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

THE PATHOGENESIS OF ACUTE APPENDICITIS. THE NON-SPECIFIC RESPONSE OF THE DIGESTIVE TRACT IN ACUTE INFLAMMATION IN THE ABDOMEN.

Eksperimental'naia i klinicheskaia gastroenterologiia = Experimental & clinical gastroenterology, 2016

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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