What is the half-life of prazosin?

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Half-Life of Prazosin

The elimination half-life of prazosin is approximately 2 to 3 hours following oral administration. 1

Pharmacokinetic Properties

The FDA-approved drug label clearly states that following oral administration, prazosin reaches peak plasma concentrations at approximately 3 hours, with a plasma half-life of 2 to 3 hours. 1 This short half-life has important clinical implications for dosing frequency and duration of action.

Key Pharmacokinetic Parameters

  • Absorption: Peak plasma levels occur between 1 and 3 hours after oral administration, with considerable interindividual variation. 2, 3

  • Bioavailability: Oral bioavailability ranges from 43.5% to 69.3% (mean 56.9%) due to extensive first-pass hepatic metabolism. 2

  • Protein Binding: Prazosin is highly bound (92-97%) to plasma proteins, specifically albumin and alpha-1-acid glycoprotein. 2, 3

  • Volume of Distribution: The steady-state volume of distribution is approximately 42.2 ± 8.9 liters. 2

  • Clearance: Total body clearance is 12.7 ± 1.3 L/h, with plasma clearance around 80 ml/min. 2

Metabolism and Elimination

  • Hepatic Metabolism: Prazosin undergoes extensive hepatic metabolism, primarily through demethylation and conjugation pathways. 1, 2

  • Excretion Routes: The drug is excreted mainly via bile and feces, with only 6% excreted unchanged in urine. 2 The two main O-demethylated metabolites are almost completely excreted in bile. 2

Clinical Implications in Special Populations

Renal Impairment

  • In chronic renal failure, the plasma elimination half-life is prolonged, the plasma free fraction increases, and peak plasma concentrations are higher. 2

  • Despite these changes, only 6% of prazosin is excreted unchanged in urine, so renal dysfunction has less impact than hepatic dysfunction. 2

Congestive Heart Failure

  • Prazosin disposition is modified in CHF patients, with increased plasma free fraction and prolonged elimination half-life. 2

  • Dosage should be titrated cautiously in these patients due to altered pharmacokinetics. 2

Hepatic Impairment

  • Given the extensive hepatic metabolism and high first-pass effect, prazosin kinetics are expected to be significantly altered in liver disease, though specific data are limited. 2

Comparison with Related Alpha-Blockers

  • Terazosin: Has a significantly longer half-life of approximately 12 hours (3-4 times longer than prazosin), allowing once-daily dosing. 4, 5

  • Prazosin's shorter half-life necessitates multiple daily dosing (typically 2-3 times daily) to maintain therapeutic blood pressure control throughout the day. 2

Important Clinical Considerations

  • The short 2-3 hour half-life means prazosin requires multiple daily doses for sustained antihypertensive effect. 1, 2

  • Despite the short half-life, tolerance has not been observed to develop during long-term therapy. 1

  • The pharmacokinetic data do not fully explain the disappearance of the first-dose hypotensive effect during continued administration, suggesting receptor-level adaptations may occur. 2

  • Considerable intrasubject consistency exists in pharmacokinetic response despite intersubject variability, meaning individual patients respond consistently to their dosing regimen. 3

References

Research

Clinical pharmacokinetics of prazosin.

Clinical pharmacokinetics, 1980

Research

Pharmacokinetics of prazosin in man.

Journal of clinical pharmacology, 1978

Research

Pharmacokinetics of terazosin.

The American journal of medicine, 1986

Research

Terazosin: a new antihypertensive agent with favorable effects on lipids.

International journal of clinical pharmacology, therapy, and toxicology, 1989

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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