What tests are used to diagnose the presence of insulin in a patient?

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Testing for the Presence of Insulin

The gold standard for diagnosing endogenous hyperinsulinism is a supervised 48-72 hour fasting test, measuring simultaneous insulin, C-peptide, and glucose levels when hypoglycemia occurs, with diagnostic criteria of insulin >3 mcIU/mL, C-peptide ≥0.6 ng/mL, and glucose <55 mg/dL. 1, 2

Diagnostic Testing Algorithm

First-Line: Supervised Fasting Test

The supervised fast remains the definitive diagnostic approach:

  • Duration: 48-72 hours under medical supervision, continuing until hypoglycemia develops or time limit reached 1, 2
  • Monitoring: Measure capillary blood glucose at regular intervals and when symptoms occur 3
  • Termination criteria: Stop when blood glucose drops to <40-45 mg/dL or patient develops neuroglycopenic symptoms (confusion, lethargy, seizures) 1

At the time of hypoglycemia, obtain simultaneous measurements of:

  • Plasma glucose
  • Serum insulin
  • Serum C-peptide
  • Proinsulin levels
  • Urine sulfonylureas (to exclude factitious hypoglycemia) 2

Diagnostic Thresholds for Endogenous Hyperinsulinism

The National Comprehensive Cancer Network establishes these criteria at the termination of the fast: 1

  • Insulin: >3 mcIU/mL (>21 pmol/L)
  • C-peptide: ≥0.6 ng/mL (≥0.2 nmol/L)
  • Proinsulin: ≥5 pmol/L
  • Glucose: <55 mg/dL (<3.0 mmol/L)
  • Insulin-to-glucose ratio: ≥0.3

Proinsulin >5 pmol/L with glucose <2.5 mmol/l achieves 100% diagnostic specificity and sensitivity for endogenous hyperinsulinism, making it the single most reliable biochemical marker. 4

Alternative: Shortened Fasting Protocols

Recent evidence supports shorter fasting tests in select cases:

  • 12-hour mini-fasting test: Performed over 3 consecutive days, this identified 100% of patients with pathologically proven endogenous hyperinsulinism when insulin ≥3 μU/ml and C-peptide ≥0.6 ng/ml occurred with symptomatic hypoglycemia 5
  • 24-hour short fasting test: Diagnosed 91.7% (11/12) of insulinoma cases within 24 hours, offering a valid first-line outpatient approach 3
  • 48-hour fasting test combined with glucagon stimulatory test: Achieved 93.3% sensitivity and 95.0% specificity, providing a less invasive alternative 6

However, the 72-hour fast remains the gold standard when shorter tests are negative but clinical suspicion persists. 5

Understanding C-Peptide vs. Insulin Measurements

C-peptide is superior to insulin for diagnosing endogenous hyperinsulinism because:

  • Produced in equimolar quantities with insulin during proinsulin cleavage 2
  • Longer half-life than insulin (30 minutes vs. 4 minutes)
  • Not extracted by the liver, unlike insulin which undergoes 50% first-pass hepatic metabolism 2
  • Distinguishes endogenous insulin production from exogenous insulin administration 7

Elevated C-peptide with elevated insulin confirms endogenous hyperinsulinism; elevated insulin with suppressed C-peptide indicates exogenous insulin administration. 7, 2

Clinical Context: When to Test

The American Diabetes Association states routine testing for insulin or proinsulin is NOT recommended in most people with diabetes or cardiovascular disease risk. 7

Testing is indicated when:

  • Whipple's triad is present: symptomatic hypoglycemia + documented low glucose (<55 mg/dL) + symptom resolution after glucose administration 1, 2
  • Differentiating type 1 from type 2 diabetes in ambiguous cases (e.g., ketoacidosis with type 2 phenotype) 7
  • Investigating suspected insulinoma or other causes of endogenous hyperinsulinism 1, 2
  • Evaluating nondiabetic hypoglycemia to rule out surreptitious insulin administration 7
  • Diagnosing islet cell tumors when inappropriately elevated insulin persists despite low glucose 7

Critical Pitfalls to Avoid

Do not rely on random insulin or C-peptide measurements: These must be obtained during documented hypoglycemia to have diagnostic value 1, 2

Ensure proper fasting conditions: For C-peptide measurement required by payers for insulin pump coverage, measure when simultaneous fasting glucose is ≤220 mg/dL (12.5 mmol/L) 7

Consider insulin resistance confounders: Isolated C-peptide elevation without hypoglycemia reflects insulin resistance (obesity, PCOS, early type 2 diabetes), not pathological hyperinsulinism 2

Avoid premature test termination: The 48-72 hour fast should continue until true hypoglycemia occurs or the time limit is reached, not stopped at arbitrary glucose thresholds 1

Test for sulfonylureas: Always check urine for sulfonylureas during the fasting test to exclude factitious hypoglycemia from oral hypoglycemic agents 2

References

Guideline

Diagnostic and Treatment Approach for Insulinoma

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Diagnostic and Management of Hyperinsulinism

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Mini-fasting tests diagnosing endogenous hyperinsulinism: a follow-up of 26 cases.

Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association, 2014

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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