In an adult with nephrotic‑range proteinuria and biopsy‑proven membranous nephropathy, what does a negative anti‑phospholipase A2 receptor (anti‑PLA2R) antibody result indicate and how should I manage it?

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Last updated: February 12, 2026View editorial policy

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False Negative Anti-PLA2R Antibody in Membranous Nephropathy

A negative anti-PLA2R antibody test in biopsy-proven membranous nephropathy requires immediate evaluation for secondary causes (malignancy, lupus, hepatitis B/C, medications) and mandates kidney biopsy tissue staining for PLA2R antigen to distinguish between immunologic remission and true seronegative disease. 1

Understanding Seronegative Membranous Nephropathy

Anti-PLA2R antibodies are detected in only 70–80% of primary membranous nephropathy cases, meaning 20–30% of patients with genuine primary disease will test negative. 1, 2 Seronegative patients fall into two distinct categories that require different management approaches:

Category 1: Recent Immunologic Remission

  • Patients may be seronegative because they have recently entered spontaneous remission, with circulating antibodies already cleared from serum. 1
  • These patients may still have PLA2R antigen detectable in glomerular immune deposits on kidney biopsy tissue immunofluorescence staining, confirming primary disease despite negative serology. 3, 1
  • This scenario represents true primary membranous nephropathy with favorable prognosis. 1

Category 2: True Seronegative Disease

  • Patients lacking both circulating anti-PLA2R antibodies and tissue PLA2R antigen have either secondary membranous nephropathy or primary disease driven by alternative antigens (THSD7A, NELL1, or yet-unidentified targets). 1, 4
  • Secondary causes are significantly more likely in this group and must be systematically excluded. 1, 4

Mandatory Evaluation for Secondary Causes

All anti-PLA2R negative patients require comprehensive screening regardless of clinical presentation: 1, 2

  • Malignancy screening: Age-appropriate cancer tests including colonoscopy, mammography, PSA, and chest imaging; consider contrast-enhanced CT chest/abdomen/pelvis in patients >60 years or with constitutional symptoms. 1, 5
  • Autoimmune evaluation: ANA, anti-dsDNA, complement levels (C3, C4) to exclude systemic lupus erythematosus. 1, 2
  • Infectious workup: Hepatitis B surface antigen, hepatitis C antibody, HIV testing. 1, 2
  • Medication review: NSAIDs, gold, penicillamine, anti-TNF agents. 1, 2

The association between malignancy and membranous nephropathy is more frequent in older patients and may be causal or coincidental, but systematic screening is essential. 1

Critical Role of Kidney Biopsy Tissue Analysis

Kidney biopsy tissue immunofluorescence staining for PLA2R antigen provides critical diagnostic information that serology cannot: 3, 4

  • Strong glomerular PLA2R staining on tissue correlates tightly with serum antibodies and confirms primary disease even when serology is negative due to recent remission. 4
  • Faint or absent PLA2R staining in glomeruli, combined with negative serology, strongly suggests secondary membranous nephropathy or alternative antigen-driven disease. 4
  • Enhanced PLA2R expression in glomeruli was found in 61 of 88 patients, with 60 of these having positive serum antibodies; the 27 seronegative patients showed only faint glomerular staining, and 15 had identifiable secondary causes. 4

The degree of chronic interstitial fibrosis and tubular atrophy on biopsy is a major adverse prognostic factor that guides whether to pursue conservative or aggressive therapy. 3

Management Algorithm for Seronegative Patients

Step 1: Confirm True Seronegativity

  • Weakly positive results require confirmation with additional assays or repeat testing to exclude false negatives. 3, 1
  • Consider immunofluorescence-based antibody testing if ELISA was initially used, as immunofluorescence is more sensitive. 3

Step 2: Tissue PLA2R Staining

  • Request immunofluorescence staining of biopsy tissue for PLA2R antigen if not already performed. 3, 1
  • Strong tissue positivity with serum negativity indicates recent immunologic remission—these patients have primary disease with excellent prognosis. 1, 4
  • Absent or faint tissue staining mandates aggressive secondary cause evaluation. 4

Step 3: Risk Stratification for Treatment

Do not initiate immunosuppression if all of the following are present: 1, 2

  • Proteinuria <3.5 g/day
  • Serum albumin >30 g/L
  • eGFR >60 mL/min/1.73 m²

Consider immunosuppression when any of these apply: 1

  • Persistent nephrotic syndrome (proteinuria ≥4 g/day, ≥50% of baseline, unchanged after 6 months of optimal RAAS blockade)
  • Severe nephrotic complications (acute kidney injury, infections, thromboembolism)
  • Declining kidney function (≥30% rise in creatinine over 6–12 months with eGFR still >25–30 mL/min/1.73 m²)

Contraindications to immunosuppression: 1

  • Serum creatinine persistently ≥3.5 mg/dL (eGFR ≤30 mL/min/1.73 m²)
  • Kidney size <8 cm on ultrasound

Step 4: Serial Monitoring Strategy

  • Repeat anti-PLA2R antibody testing every 3–6 months even if initially negative, as seroconversion can occur and predicts disease progression. 1, 6
  • Rising antibody titers forecast disease relapse before proteinuria increases, allowing preemptive therapeutic adjustments. 1, 6
  • Monitor proteinuria, serum albumin, and eGFR every 3 months during observation period. 1

First-Line Immunosuppressive Options (When Indicated)

When treatment criteria are met, choose among: 1

  • Rituximab (preferred in many centers due to favorable safety profile)
  • Cyclophosphamide with alternating monthly glucocorticoids for 6 months
  • Tacrolimus-based therapy for ≥6 months

Critical Pitfalls to Avoid

  • Do not assume negative PLA2R serology excludes primary disease—tissue staining may reveal recent immunologic remission. 3, 1
  • Do not skip secondary cause evaluation—even positive PLA2R antibodies do not exclude concurrent malignancy, lupus, or hepatitis. 1, 5, 2
  • Do not use THSD7A or NELL1 antibodies alone for diagnosis—these lack the diagnostic accuracy of PLA2R testing and require biopsy confirmation. 3, 2
  • Do not delay biopsy when eGFR is rapidly declining—this suggests alternative or additional pathology requiring histologic evaluation. 3, 2
  • Do not base treatment decisions on single antibody measurements—serial trends provide far more reliable guidance than isolated values. 1, 2
  • Do not ignore mesangial deposits on electron microscopy—these occur in 27.5% of PLA2R-positive cases and do not reliably distinguish primary from secondary disease. 7

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Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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