What work‑up should be performed for a child with chronic diarrhea lasting at least two weeks?

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Work-up for Chronic Diarrhea in Children

Initial Clinical Evaluation

Begin with a focused history targeting age-specific etiologies, stool characteristics, and red-flag symptoms to guide your diagnostic algorithm. 1

Critical History Elements

  • Age-specific considerations: Neonates and infants <6 months warrant investigation for congenital enteropathies, anatomic abnormalities (e.g., malrotation), and genetic disorders, while older children more commonly have infectious, allergic, or immune-mediated causes 1, 2

  • Stool characteristics:

    • Watery diarrhea suggests osmotic (carbohydrate malabsorption, laxative use) or secretory mechanisms (bile acid malabsorption, microscopic colitis) 3
    • Fatty/greasy stools (steatorrhea) with weight loss indicates malabsorption (celiac disease, giardiasis, pancreatic insufficiency) 3
    • Bloody or mucoid stools point to inflammatory bowel disease, invasive bacterial/parasitic infections, or cow's milk protein allergy 3, 1
  • Red-flag symptoms requiring urgent evaluation: Severe weight loss or failure to thrive, fever, bloody diarrhea, severe abdominal pain, signs of malnutrition (edema, muscle wasting), or family history of inflammatory bowel disease or immunodeficiency 1

Physical Examination Priorities

  • Growth parameters: Plot weight, height, and weight-for-height on growth charts to identify faltering growth or malnutrition 1
  • Signs of malabsorption: Muscle wasting, edema, pallor (anemia), dermatitis, glossitis, or clubbing 1
  • Abdominal examination: Distension, organomegaly, masses, or perianal disease (fissures, fistulas, skin tags suggesting Crohn disease) 1

First-Tier Laboratory Investigations

Order these initial tests for all children with chronic diarrhea lasting ≥2 weeks to categorize the mechanism and identify common treatable causes. 1, 4

Stool Studies

  • Stool microscopy and culture: Identify bacterial pathogens (Salmonella, Shigella, Campylobacter, pathogenic E. coli) and parasites (Giardia, Cryptosporidium, Entamoeba histolytica) 1, 4
  • Fecal calprotectin: Elevated levels (>50 µg/g) indicate intestinal inflammation and help distinguish inflammatory bowel disease from functional disorders 3
  • Stool pH and reducing substances: pH <6.0 and reducing substances >0.5% suggest carbohydrate malabsorption, though these findings alone do not mandate dietary changes 5, 4
  • Fecal fat (72-hour collection or spot Sudan stain): Quantify steatorrhea to confirm malabsorption; >7 g/day (or >15% of fat intake) is abnormal 4
  • Fecal elastase-1: Low levels (<200 µg/g) indicate pancreatic insufficiency 1

Blood Tests

  • Complete blood count with differential: Anemia (iron, B12, or folate deficiency from malabsorption), eosinophilia (parasitic infection, allergic enteropathy), or leukocytosis (infection, inflammation) 1
  • Inflammatory markers: Elevated ESR or CRP support inflammatory bowel disease or chronic infection 1
  • Celiac serology: Tissue transglutaminase IgA (tTG-IgA) with total IgA level; if IgA-deficient, order tTG-IgG or deamidated gliadin peptide antibodies 1, 3
  • Electrolytes, albumin, and total protein: Hypoalbuminemia and hypoproteinemia indicate protein-losing enteropathy; electrolyte abnormalities reflect secretory losses 1, 4
  • Thyroid function tests: Hyperthyroidism can cause chronic diarrhea in older children 3

Second-Tier Investigations (Based on Initial Results)

Proceed to these tests when first-tier studies are unrevealing or when specific etiologies are suspected. 1

Endoscopy with Biopsy

  • Upper endoscopy (esophagogastroduodenoscopy) with duodenal biopsies: Indicated for suspected celiac disease (even with negative serology if clinical suspicion is high), giardiasis, or small bowel mucosal disorders (villous atrophy, microvillus inclusion disease) 1, 3
  • Colonoscopy with biopsies: Perform when fecal calprotectin is elevated, bloody diarrhea is present, or inflammatory bowel disease is suspected; biopsies can also diagnose microscopic colitis or allergic colitis 1, 3

Imaging Studies

  • Abdominal ultrasound: First-line imaging to detect anatomic abnormalities (malrotation, intussusception), bowel wall thickening, or organomegaly 2, 1
  • Upper GI series with small bowel follow-through or MR enterography: Evaluate for malrotation, strictures, fistulas, or Crohn disease when inflammatory bowel disease is suspected 1

Specialized Tests

  • Hydrogen breath tests: Diagnose lactose intolerance, fructose malabsorption, or small intestinal bacterial overgrowth (SIBO) 1, 3
  • Sweat chloride test: Rule out cystic fibrosis in children with steatorrhea, failure to thrive, or recurrent respiratory infections 1
  • Immunodeficiency screening: Measure immunoglobulin levels (IgA, IgG, IgM, IgE), lymphocyte subsets, and HIV testing if recurrent infections, severe diarrhea, or poor growth suggest immune dysfunction 1

Third-Tier Investigations (Refractory or Complex Cases)

Reserve these for children with persistent diarrhea despite initial management or when congenital/genetic disorders are suspected. 1

Genetic Testing

  • Targeted gene panels or whole-exome sequencing: Indicated for neonates/infants with severe, intractable diarrhea suggesting congenital diarrheal disorders (e.g., microvillus inclusion disease, tufting enteropathy), monogenic inflammatory bowel disease, or primary immunodeficiency 1

Advanced Functional Studies

  • Intestinal permeability tests (lactulose/mannitol ratio): Assess mucosal barrier function in suspected enteropathy 4
  • Radionuclide imaging (e.g., SeHCAT scan): Quantify bile acid malabsorption when secretory diarrhea persists without clear cause 1, 3

Common Pitfalls to Avoid

  • Do not delay referral to pediatric gastroenterology for infants <6 months with chronic diarrhea, children with severe malnutrition, or those with suspected inflammatory bowel disease or congenital enteropathy—early specialist involvement minimizes morbidity 1
  • Do not empirically restrict lactose or other nutrients based solely on low stool pH or reducing substances; true lactose intolerance requires clinical correlation (severe diarrhea upon reintroduction) 5
  • Do not order stool cultures for chronic diarrhea without acute onset or travel history unless parasitic infection is suspected; most bacterial pathogens cause acute, not chronic, diarrhea 1
  • Do not overlook cow's milk protein allergy in infants with bloody diarrhea and failure to thrive; a 2–4 week trial of extensively hydrolyzed or amino acid-based formula is both diagnostic and therapeutic 1
  • Do not perform endoscopy before checking celiac serology unless the child is already on a gluten-free diet, as serology is less invasive and highly sensitive 1, 3

Algorithmic Summary

  1. Categorize by stool type (watery, fatty, bloody) and age to prioritize differential diagnoses 1, 3
  2. Order first-tier tests (stool studies, CBC, celiac serology, albumin, fecal calprotectin) for all patients 1, 4
  3. Proceed to endoscopy if inflammatory markers or fecal calprotectin are elevated, or if malabsorption is confirmed without clear cause 1, 3
  4. Consider imaging (ultrasound, upper GI series) if anatomic abnormalities or Crohn disease are suspected 2, 1
  5. Refer for genetic testing in neonates/infants with severe, refractory diarrhea or when congenital enteropathy is suspected 1

References

Research

Approach to a Child with Chronic Diarrhea.

Indian journal of pediatrics, 2024

Research

Evaluation of chronic diarrhea.

American family physician, 2011

Research

Investigation of chronic diarrhoea in infancy.

Early human development, 2013

Guideline

Management of Pediatric Diarrhea with Dehydration and Electrolyte Disturbances

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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