What is Acute Inflammatory Demyelinating Polyneuropathy (AIDP)?

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From the Guidelines

Definition of Acute Inflammatory Demyelinating Polyneuropathy (AIDP)

Acute inflammatory demyelinating polyneuropathy (AIDP), also known as Guillain-Barré syndrome, is a rare autoimmune disorder characterized by rapid onset of muscle weakness and paralysis.

  • It is an inflammatory disease of the peripheral nervous system (PNS) and is the most common cause of acute flaccid paralysis, with an annual global incidence of approximately 1–2 per 100,000 person-years 1.
  • AIDP is thought to be caused by an aberrant immune response to infections that results in damage to peripheral nerves, although the pathogenesis is not fully understood 1.
  • The clinical presentation of AIDP is heterogeneous, and several distinct clinical variants exist, including a pure motor variant, paraparetic variants, and Miller Fisher syndrome (MFS) 1.

Diagnosis and Treatment

  • Diagnosis of AIDP is based on the patient history and neurological, electrophysiological, and cerebrospinal fluid (CSF) examinations 1.
  • Treatment typically involves plasma exchange or intravenous immunoglobulin (IVIG) at a dose of 2g/kg administered over 2-5 days, with corticosteroids such as prednisone 60mg daily for 1-2 weeks also considered in some cases 1.
  • Supportive care, including pain management with medications like gabapentin 300-1200mg daily and physical therapy, is also essential to manage symptoms and promote recovery.
  • In severe cases, prompt recognition of symptoms is essential to prevent respiratory insufficiency due to affected cervical nerve roots, and treatment may involve IVIG and/or plasma exchange or selective separation 1.

Clinical Features and Outcome

  • The severity of AIDP is highly variable, ranging from mild distal limb weakness to complete paralysis, respiratory failure, and even death 1.
  • About 20% of patients with AIDP develop respiratory failure and require mechanical ventilation, and cardiac arrhythmias and blood pressure instability can occur owing to involvement of the autonomic nervous system 1.
  • The outcome of AIDP is generally favorable, with 60–80% of patients able to walk independently 6 months after disease onset, with or without treatment 1.

From the Research

Definition and Characteristics of Acute Inflammatory Demyelinating Polyneuropathy (AIDP)

  • AIDP is characterized by the autoimmune destruction of Schwann cells with resultant myelin degradation 2
  • It is the most common subtype of Guillain-Barré Syndrome (GBS) 2, 3, 4
  • AIDP is an immune-mediated polyneuropathy usually triggered by infections or vaccinations 3

Clinical Presentation

  • Symptom onset is typically 1-3 weeks following an upper respiratory or gastrointestinal illness 2
  • Rapidly progressive ascending areflexic motor paralysis is a common symptom 2, 5
  • Lower cranial nerves are often involved, leading to bulbar weakness and respiratory compromise 2
  • Autonomic dysregulation is common and must be managed carefully to avoid potentially fatal autonomic dysregulation 2
  • Initial complaint can be lower back pain in up to 66% of GBS cases 2

Diagnosis and Treatment

  • Diagnosis is largely clinical, but diagnostic tools are available to confirm the diagnosis, prognosticate, detect variant forms, and rule out mimics 5
  • Intravenous Immunoglobulin (IVIg) and/or plasmapheresis coupled with supportive management is the mainstay of GBS treatment 2, 3, 4
  • The majority of patients make a full recovery in up to one year 2, 3

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Acute inflammatory demyelinating polyradiculoneuropathy in a newborn infant.

European journal of paediatric neurology : EJPN : official journal of the European Paediatric Neurology Society, 2016

Research

Acute/chronic inflammatory polyradiculoneuropathy.

Handbook of clinical neurology, 2023

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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