Does Tetrahydrocannabinol (THC) prolong the QT interval?

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Last updated: May 17, 2025View editorial policy

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From the Guidelines

THC is not generally considered a QT-prolonging drug, and current evidence does not suggest it directly causes significant QT interval prolongation. When considering the management of patients with ventricular arrhythmias and the prevention of sudden cardiac death, as outlined in the 2017 AHA/ACC/HRS guideline 1, the focus is on avoiding QT-prolonging medications in patients with long QT syndrome unless no alternative is available. While this guideline highlights the importance of monitoring the QTc interval during therapy with QT-prolonging medications, it does not specifically mention THC as a QT-prolonging agent.

The primary concern with THC and cardiac function relates more to its effects on the autonomic nervous system and cannabinoid receptors in the heart and blood vessels, which can lead to mild tachycardia and changes in blood pressure. However, these effects do not typically result in significant QT interval prolongation. For patients with pre-existing heart conditions or those taking medications known to prolong the QT interval, caution is advised due to potential indirect effects on cardiac function.

Key points to consider include:

  • THC's impact on the autonomic nervous system and cannabinoid receptors
  • Potential for mild tachycardia and changes in blood pressure
  • Importance of discussing cannabis use with healthcare providers, especially for those with cardiac risk factors or taking other medications
  • The need for careful monitoring in patients with long QT syndrome, as outlined in the guideline 1, although THC is not specifically identified as a risk factor.

From the Research

QT Prolonging Drugs

  • The QT interval represents ventricular depolarisation and repolarisation, and prolongation of this interval can lead to life-threatening complications, including arrhythmias such as Torsades de Pointes and Ventricular Fibrillation 2.
  • Many drug therapies are associated with prolongation of the QT interval, which may increase the risk of Torsades de Pointes (TdP), a potentially life-threatening cardiac arrhythmia 3.
  • Non-cardiac drugs, such as antipsychotics, antidepressants, and macrolides, may prolong the QT interval, especially in case of overdosage or pharmacokinetic interactions 4.

Risk Factors for QT Prolongation

  • Patient-related risk factors, such as female sex, age >65 years, and uncorrected electrolyte disturbances, can increase the risk of QT prolongation 3.
  • Co-prescribed medicines that could increase the risk of QT prolongation, such as azole antifungals, macrolide antibacterials, and class-I or -III antiarrhythmic agents, should be considered 4.
  • Other factors influencing QT interval, such as hypokalemia, hypomagnesemia, bradycardia, increased age, female sex, congestive heart failure, and polymorphisms of genes coding ion channels or enzymes involved in drug metabolism, should be taken into account 5.

THC and QT Prolongation

  • There is no direct evidence in the provided studies to suggest that THC is a QT prolonging drug 3, 6, 2, 4, 5.
  • The studies provided focus on the QT prolonging effects of various drugs, including antipsychotics, antidepressants, and macrolides, but do not mention THC specifically.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Antipsychotic Medication and QT Prolongation.

Pakistan journal of medical sciences, 2015

Research

Managing drug-induced QT prolongation in clinical practice.

Postgraduate medical journal, 2021

Research

Antipsychotic drugs and QT prolongation.

International clinical psychopharmacology, 2005

Research

Drug- and non-drug-associated QT interval prolongation.

British journal of clinical pharmacology, 2010

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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