What is the mechanism of Sodium-Glucose Linked Transporter 2 Inhibitors (SGLT2i)?

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From the Guidelines

Sodium-Glucose Linked Transporter 2 Inhibitors (SGLT2i) work by blocking the reabsorption of glucose in the proximal renal tubule, increasing glucose elimination and reducing hyperglycemia.

Mechanism of Action

  • SGLT2 proteins are expressed in the proximal convoluted tubule of the kidneys, where they play a key role in glucose homeostasis 1.
  • SGLT2 inhibitors block the resorption of glucose by the proximal renal tubules, increasing glucose elimination and reducing hyperglycemia 1.
  • This effect is more pronounced in the setting of hyperglycemia, where significant amounts of glucose are filtered into the urine 1.
  • Beyond their effect on blood glucose, SGLT2 inhibitors also cause diuretic and natriuretic effects, weight loss, and lowering of systolic blood pressure 1.

Key Benefits

  • SGLT2 inhibitors provide insulin-independent glucose lowering, making them a useful treatment option for patients with type 2 diabetes 1.
  • They also provide modest weight loss and blood pressure reduction, which can be beneficial for patients with cardiovascular risk factors 1.
  • SGLT2 inhibitors have been shown to reduce the risk of major adverse cardiovascular events (MACE) and hospitalization for heart failure (HF) in patients with type 2 diabetes and high cardiovascular risk 1.

From the Research

Mechanism of Action

The mechanism of Sodium-Glucose Linked Transporter 2 Inhibitors (SGLT2i) involves the inhibition of renal glucose reabsorption by blocking the SGLT2 cotransporters in the proximal tubules, causing glucosuria 2. This leads to a reduction in glycemia and lowers HbA1c by approximately 1.0% 2.

Key Effects

Some key effects of SGLT2i include:

  • Reduction in intraglomerular pressure, which contributes to the nephroprotective effects of SGLT2i 2
  • Caloric loss, which reduces weight, increases insulin sensitivity, and improves lipid metabolism 2
  • Metabolic shifts towards gluconeogenesis and ketogenesis, thought to be protective for the heart and kidneys 2, 3
  • Reduction in tubular cell glucotoxicity through reduced mitochondrial dysfunction and inflammation 2
  • Improvement in blood pressure through a negative sodium and water balance and possibly by inhibiting the sympathetic nervous system 2, 4

Cardiovascular and Renal Benefits

SGLT2i have been shown to have significant benefits in renal and cardiovascular protection, including:

  • Reduction in cardiovascular events such as heart failure 4, 3
  • Renal protection, including reduced glomerular hyperfiltration and albuminuria 3, 5
  • Improved cardiovascular function, potentially due to reduced volume retention and blood pressure, and preserved heart function 5

Additional Mechanisms

Other possible mechanisms of SGLT2i include:

  • Reduction in inflammatory markers, fibrosis, and podocyte injury 2
  • Stimulation of erythropoiesis, which improves organ oxygen delivery 5
  • Activation of the tubule-glomerular feedback, contributing to favorable outcomes 4

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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