What is the metabolism of paliperidone (Invega)?

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Metabolism of Paliperidone (Invega)

Primary Metabolic Pathway

  • Paliperidone undergoes minimal hepatic metabolism with renal excretion being the major route of elimination. 1
  • Paliperidone is the active metabolite of risperidone (9-hydroxyrisperidone), but has a distinctly different metabolic profile from its parent compound 2, 3
  • Unlike many antipsychotics, paliperidone is not extensively metabolized by the cytochrome P450 (CYP) enzyme system 1, 4

Limited Hepatic Metabolism

  • Paliperidone has four minor metabolic pathways, but these represent only a small fraction of its total body clearance 3
  • In vitro studies have shown that CYP2D6 and CYP3A4 may be minimally involved in paliperidone metabolism, but in vivo studies do not show significant elimination by these isozymes 1
  • Paliperidone is not a substrate of CYP1A2, CYP2A6, CYP2C9, and CYP2C19 enzymes 1
  • P-glycoprotein (P-gp) has been identified as a transporter for paliperidone, with paliperidone being a P-gp substrate 1, 5

Excretion Profile

  • Renal excretion is the predominant elimination pathway for paliperidone, with approximately 59% of the dose excreted unchanged in urine 2, 3
  • The terminal elimination half-life of paliperidone is approximately 23 hours 2
  • Pharmacokinetics are dose-proportional within the recommended dose range 2

Clinical Implications of Metabolic Profile

  • Being a CYP2D6 poor metabolizer is not clinically relevant for paliperidone treatment, unlike with risperidone therapy 3
  • Paliperidone has a lower potential for drug-drug interactions compared to many other antipsychotics due to its limited hepatic metabolism 5
  • Dose adjustment is necessary in patients with renal impairment since renal excretion is the major elimination pathway 1, 5

Drug Interactions

  • Paliperidone does not substantially inhibit drugs metabolized by cytochrome P450 isozymes, including CYP1A2, CYP2A6, CYP2C8/9/10, CYP2D6, CYP2E1, CYP3A4, and CYP3A5 1
  • Strong CYP3A4 and P-gp inducers like carbamazepine can decrease paliperidone plasma concentrations by approximately 37% through increased renal clearance 1
  • Valproate coadministration may increase paliperidone exposure by approximately 50%, potentially requiring dose reduction 1
  • Paliperidone is a weak inhibitor of P-glycoprotein (P-gp) at high concentrations, but the clinical relevance is unknown 1

Comparison to Risperidone

  • Unlike risperidone, which is extensively metabolized by CYP2D6 to form paliperidone, paliperidone itself undergoes minimal hepatic metabolism 3, 4
  • Paliperidone may be preferred over risperidone when liver disease, drug-drug interactions, or other alterations in metabolism make appropriate dosing of risperidone difficult 2
  • Paliperidone has decreased inhibition of P-glycoprotein compared to risperidone, further reducing potential for drug-drug interactions 5

Special Populations

  • Patients with renal impairment require dose adjustments due to the predominant renal elimination pathway 1, 6
  • Hepatic impairment has minimal impact on paliperidone pharmacokinetics due to its limited hepatic metabolism 5

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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