What are the typical lab results in hypoaldosteronism?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: November 27, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Laboratory Results in Hypoaldosteronism

Hypoaldosteronism characteristically presents with hyperkalemia (>94% of cases), often accompanied by hyponatremia (54.5% of cases) and metabolic acidosis (60.3% of cases), with low plasma aldosterone and low or inappropriately normal plasma renin activity. 1

Core Laboratory Abnormalities

Electrolyte Disturbances

  • Hyperkalemia is the hallmark finding, present in 94.6% of cases, with mean serum potassium of 5.4 ± 0.5 mmol/L 1
  • Hyponatremia occurs in approximately 54.5% of cases (mean serum sodium: 132.1 ± 6.3 mmol/L), particularly hypovolemic hyponatremia due to urinary sodium wasting 1
  • Hyperchloremic metabolic acidosis is present in 60.3% of cases (mean serum bicarbonate: 22.6 ± 3.3 mmol/L) due to impaired H+ excretion in the distal nephron 1, 2

Hormonal Profile

  • Low plasma aldosterone concentration is the defining feature, with baseline levels typically ranging from 5.4 to 21.6 ng/dL 3
  • Suppressed plasma renin activity (0.12 to 1.3 ng/mL/hr) is characteristic of the most common form—hyporeninemic hypoaldosteronism—occurring in approximately 70% of adult cases 4, 3
  • Normal cortisol reserve distinguishes isolated hypoaldosteronism from primary adrenal insufficiency, which occurs in only 5% of hypoaldosteronism cases 1, 3

Diagnostic Approach

Key Distinguishing Features

  • The stimulated plasma aldosterone/serum potassium ratio below 3 is highly useful for diagnosis, as stimulated aldosterone reflects bioactivity on the collecting tubule 4
  • Unlike primary aldosteronism where plasma aldosterone should be at least 10-15 ng/dL for a positive test 5, hypoaldosteronism shows inappropriately low aldosterone relative to the elevated potassium
  • Urinary electrolyte patterns show increased urinary sodium excretion (sodium wasting) and decreased urinary potassium excretion 1, 2

Clinical Context

  • Patients are typically older (median age 77 years, often >50 years) with diabetes mellitus and/or nephropathy 1, 3
  • Mild to moderate renal insufficiency is common and contributes to the severity of hyperkalemia and acidosis 2, 3
  • Hypovolemia, when present, is associated with more florid clinical presentation and more severe hyponatremia 1

Important Caveats

  • Do not rely on hyperkalemia alone: While present in >94% of cases, approximately 25% of patients may have normokalemia at presentation 4
  • Hyponatremia is not universal: Only about half of patients demonstrate sodium depletion, so its absence does not exclude the diagnosis 1, 4
  • Consider medication effects: Prostaglandin inhibitors, beta-blockers, ACE inhibitors, and potassium-sparing diuretics can contribute to or mimic hypoaldosteronism 2
  • Exclude primary adrenal insufficiency: Check cortisol reserve and ACTH levels, as the combination of hyponatremia and hyperkalemia occurs in only about 50% of primary adrenal insufficiency cases at diagnosis 6

References

Research

[Hyporeninemic hypoaldosteronism and the differential diagnosis of hyperkalemia].

Schweizerische medizinische Wochenschrift, 1982

Research

Diagnostic value of plasma aldosterone/potassium ratio in hypoaldosteronism.

Journal of the Formosan Medical Association = Taiwan yi zhi, 1995

Guideline

Screening for Primary Aldosteronism

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.