What is the class and mechanism of action of Teicoplanin (glycopeptide antibiotic)?

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Teicoplanin: Class and Mechanism of Action

Teicoplanin is a glycopeptide antibiotic that inhibits bacterial cell wall biosynthesis by binding to the D-alanyl-D-alanine terminus of peptidoglycan precursors, preventing cross-linking and cell wall assembly. 1, 2, 3

Antibiotic Class

  • Teicoplanin belongs to the glycopeptide class of antibiotics, structurally related to vancomycin but with distinct chemical properties 1, 2
  • It is a fermentation product of Actinoplanes teicomyceticus, isolated in the mid-1970s 2
  • The molecule consists of six fatty-acid components attached to a common aglycone, making it 50 to 100 times more lipophilic than vancomycin 2

Mechanism of Action

  • Teicoplanin achieves its bactericidal effect by binding to acyl-D-alanyl-D-alanine residues in the bacterial cell wall, the same target as vancomycin 3, 4
  • This binding physically blocks the transglycosylation and transpeptidation reactions necessary for peptidoglycan synthesis 2, 3
  • By preventing cell wall cross-linking, teicoplanin causes bacterial cell lysis and death in a concentration-dependent manner 5

Antimicrobial Spectrum

  • Activity is restricted exclusively to Gram-positive aerobic and anaerobic bacteria; teicoplanin has no activity against Gram-negative organisms 6, 3, 7
  • The spectrum includes staphylococci (including methicillin-resistant Staphylococcus aureus [MRSA]), streptococci, corynebacterium, listeria, and anaerobic cocci 2, 3
  • Teicoplanin is generally more active than vancomycin against streptococci and Gram-positive anaerobes, exhibits similar activity against S. aureus, but vancomycin may be more active against some coagulase-negative staphylococci strains 3, 7

Clinical Context and Resistance Considerations

  • Teicoplanin became a first-choice antibiotic for severe hospital-acquired Gram-positive infections, particularly due to the pandemic of MRSA 1
  • The Centers for Disease Control and Prevention emphasizes judicious use to prevent emergence of resistant organisms like vancomycin-resistant enterococci (VRE) 6
  • Resistance to teicoplanin is difficult to induce under laboratory conditions, though it has been reported with coagulase-negative staphylococci 2, 3
  • Cross-resistance between teicoplanin and vancomycin can occur through various mechanisms, particularly among enterococci and S. aureus 5

Important Caveats

  • When broad-spectrum coverage is needed, teicoplanin must be combined with other antimicrobials since it lacks Gram-negative activity 6
  • Inoculum size and culture media composition can influence teicoplanin's in vitro activity, particularly against coagulase-negative staphylococci 3
  • The drug's high lipophilicity (50-100 times greater than vancomycin) affects its tissue distribution and allows for intramuscular administration 2, 7

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Microbiological properties of teicoplanin.

The Journal of antimicrobial chemotherapy, 1988

Guideline

Teicoplanin Use in ICU Patients

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Teicoplanin.

The Medical clinics of North America, 1995

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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