Which medication is more likely to cause parkinsonism and dyskinesia at low doses, Ziprasidone or Amisulpride?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: January 10, 2026View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Amisulprida apresenta maior risco de parkinsonismo e discinesia em doses baixas comparada à Ziprasidona

A amisulprida causa sintomas extrapiramidais, incluindo parkinsonismo e discinesia, mesmo em doses baixas, enquanto a ziprasidona geralmente requer doses mais altas para manifestar esses efeitos. 1

Evidência Específica sobre Amisulprida em Doses Baixas

  • Relatos de caso documentam que a amisulprida causa sintomas extrapiramidais (acatisia, distonia aguda e parkinsonismo induzido por drogas) mesmo em doses baixas, contrariando a alegação de que sua seletividade límbica e menor ligação aos receptores dopaminérgicos estriatais resultariam em baixa incidência de SEP 1

  • A amisulprida demonstrou ser mais eficaz que a ziprasidona, mas essa eficácia superior pode estar associada a maior bloqueio dopaminérgico D2, o que explica o risco aumentado de sintomas extrapiramidais 2

Perfil de Sintomas Extrapiramidais da Ziprasidona

  • A ziprasidona está classificada em posição intermediária no ranking de risco de SEP: clozapina < quetiapina < olanzapina = ziprasidona, com risco geralmente manifestando-se em doses mais altas 3

  • No estudo EUFEST com pacientes de primeiro episódio, a ziprasidona causou aumento de acatisia (+14% após um mês), mas o parkinsonismo foi menos frequente comparado ao haloperidol 4

  • A ziprasidona produz ligeiramente mais sintomas extrapiramidais que a olanzapina, mas os efeitos são geralmente dose-dependentes 2

Mecanismo Farmacológico Explicativo

  • O risco de SEP está inversamente relacionado à potência antidopaminérgica (receptor D2): quanto maior a ligação ao D2, maior o risco de parkinsonismo e discinesia 3

  • A alta potência antiserotonérgica (receptor 5-HT2A) da ziprasidona pode limitar os SEP, oferecendo proteção relativa em doses terapêuticas 3

  • A amisulprida tem pouca ou nenhuma ação em receptores serotonérgicos, dependendo exclusivamente de sua "seletividade límbica" teórica, que na prática clínica não previne SEP em doses baixas 1

Implicações Clínicas Práticas

  • Para pacientes com alto risco de sintomas extrapiramidais (jovens do sexo masculino, idosos), a ziprasidona seria preferível à amisulprida, especialmente se doses baixas forem necessárias 5, 6

  • Monitore com a escala AIMS (Abnormal Involuntary Movement Scale) a cada 3-6 meses em ambos os medicamentos, mas mantenha vigilância especial com amisulprida mesmo em doses baixas 5

  • Se surgirem SEP com amisulprida em dose baixa, a primeira estratégia é trocar para antipsicótico atípico com menor risco (quetiapina, clozapina), não apenas reduzir a dose 6

Armadilha Clínica Importante

  • Não assuma que "dose baixa" de amisulprida protege contra SEP - os relatos de caso demonstram claramente que isso não é verdade na prática clínica 1

  • A ziprasidona pode causar distonia aguda, parkinsonismo e acatisia grave, mas tipicamente em doses de 80 mg/dia ou superiores, não em doses iniciais 7

References

Research

Extrapyramidal side effects with low doses of amisulpride.

Indian journal of psychiatry, 2014

Research

Ziprasidone versus other atypical antipsychotics for schizophrenia.

The Cochrane database of systematic reviews, 2009

Research

Extrapyramidal symptoms during treatment of first schizophrenia episode: results from EUFEST.

European neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology, 2014

Guideline

Differentiating Extrapyramidal Symptoms (EPS) and Tardive Dyskinesia

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Extrapyramidal Symptoms: Causes, Risk Factors, and Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Research

Acute dystonia, akathisia, and parkinsonism induced by ziprasidone.

American journal of therapeutics, 2014

Related Questions

Can amisulpride cause brain tumors?
What are the management options for severe akathisia and vomiting associated with amisulpride (antipsychotic) therapy?
What antipsychotics are least likely to cause acute dystonia?
How to taper off amisulpride (antipsychotic medication)?
What is the comparative risk of extrapyramidal symptoms (EPS) between Risperidone (risperidone) and Olanzapine (olanzapine)?
To what extent does escitalopram (Selective Serotonin Reuptake Inhibitor - SSRI) depend on CYP3A4 (Cytochrome P450 3A4) for its metabolism?
What is the treatment for an adult or adolescent patient with no underlying medical conditions and a distal fibula fracture in 2 places?
What is a massive transfusion protocol in cases of severe bleeding?
What is the most likely diagnosis for a patient with a history of Atrial Septal Defect (ASD), hypertension, and hypothyroidism, who presents with severe chest pain (9/10), tachycardia, hypertension, and an Electrocardiogram (EKG) showing a Right Bundle Branch Block (RBBB) pattern, ST segment depression, and Q waves, after experiencing recurrent episodes of dizziness and a recent syncopal episode?
What alternative treatments can be used for a patient with a history of bipolar disorder, previously treated with Adderall (amphetamine and dextroamphetamine) for increased energy, who may experience sedation with quetiapine (Seroquel)?
What is the best course of treatment for a postpartum patient with persistent tingling and buzzing sensations, possibly related to vitamin D deficiency, without joint pain or swelling, that has not improved after a month?

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.