What are the risk factors for developing Post Infectious Glomerulonephritis (infection-related kidney inflammation), particularly in children and young adults with a history of streptococcal infections, such as strep throat or skin infections?

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Risk Factors for Post-Infectious Glomerulonephritis

The primary risk factors for developing post-infectious glomerulonephritis include young age (particularly children aged 2-14 years), recent streptococcal pharyngitis or skin infection (impetigo), and exposure to nephritogenic strains of Group A beta-hemolytic streptococcus, with the disease occurring after a latent period of 1-3 weeks following the initial infection. 1, 2, 3

Age-Related Risk Factors

Children represent the highest-risk population, with peak incidence occurring between ages 2-10 years 4:

  • Highest carriage rates of Group A streptococcus occur in children aged ≤14 years (10.9%), compared to 2.3% in adults aged 15-44 years and only 0.6% in those ≥45 years 1
  • School-aged children (6-14 years) demonstrate the highest rates of asymptomatic streptococcal carriage, reaching up to 26% in some populations 1
  • The epidemiology has shifted in recent decades, with adults (particularly elderly and immunocompromised individuals) now representing a significant proportion of cases in high-income countries 5

Infectious Risk Factors

Streptococcal Infections

Group A beta-hemolytic streptococcal infections are the classic trigger, with specific characteristics 1, 2, 3:

  • Pharyngitis (strep throat) typically precedes glomerulonephritis by 1-2 weeks 6
  • Skin infections (impetigo) have a longer latency period of 4-6 weeks before nephritis develops 6
  • Exposure to nephritogenic strains containing specific antigens (nephritis-associated plasmin receptor [NAPlr] and streptococcal pyrogenic exotoxin B [SPeB]) increases risk 2

Non-Streptococcal Infections (Emerging Risk)

Non-streptococcal organisms are increasingly recognized as causative agents, particularly in adults 2, 5:

  • Staphylococcal infections (skin, endocarditis, deep-seated infections) are now the main etiological agents in high-income countries among adults 5
  • Group C and G beta-hemolytic streptococci can cause acute glomerulonephritis, though a clear causal relationship remains less established 1

Host-Related Risk Factors

Immunologic Factors

  • Asymptomatic pharyngeal carriers of Group A streptococci have an extremely low risk of post-streptococcal complications 1
  • Immunocompromised status increases susceptibility, particularly in adults 5

Comorbid Conditions (Adult-Specific)

Adults with certain comorbidities face higher risk and worse outcomes 5:

  • Diabetes mellitus increases risk and is associated with failure to recover renal function 5
  • Elderly patients demonstrate higher susceptibility and poorer prognosis 5

Environmental and Geographic Risk Factors

Socioeconomic factors significantly influence disease incidence 3:

  • Poor access to clean drinking water, inadequate sanitation, and suboptimal hygiene increase risk 3
  • The incidence is declining in developed countries but remains a significant public health problem in low- and middle-income countries 3
  • Epidemic forms of the disease occur in settings with crowding and poor hygiene 7

Laboratory Markers Associated with Severity

While not traditional "risk factors" for developing the disease, certain laboratory findings at presentation predict worse renal outcomes 8:

  • Decreased C4 complement levels (OR 11.53) strongly predict decreased GFR 8
  • Hypoalbuminemia (OR 4.83) is associated with more severe renal involvement 8
  • Elevated inflammatory markers (CRP >normal, OR 3.58; elevated WBC, neutrophil count, and neutrophil/lymphocyte ratio) correlate with decreased GFR 8

Clinical Pitfalls to Avoid

  • Do not assume asymptomatic streptococcal carriers are at high risk—they have extremely low risk of developing post-streptococcal complications 1
  • In adults presenting with infection-related glomerulonephritis, consider non-streptococcal organisms (especially Staphylococcus) as the causative agent, particularly if there is concurrent endocarditis, skin infection, or other deep-seated infection 5
  • History of rheumatic fever places patients at higher risk for complications from Group A streptococcal pharyngitis, warranting more aggressive prevention strategies 1

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Post-infectious glomerulonephritis.

Paediatrics and international child health, 2017

Research

Acute post-streptococcal glomerulonephritis in children - treatment standard.

Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association, 2025

Research

[Postinfectious acute glomerulonephritis].

Nephrologie & therapeutique, 2009

Guideline

Management of Post-Streptococcal Glomerulonephritis (PSGN)

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

Prognosis of Post-Infectious Glomerulonephritis

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Research

Pediatric post-streptococcal glomerulonephritis: Clinical and laboratory data.

Pediatrics international : official journal of the Japan Pediatric Society, 2018

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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