What are the classification criteria for diabetic ketoacidosis (DKA)?

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Classification of Diabetic Ketoacidosis (DKA)

DKA is diagnosed when all three core criteria are simultaneously present: blood glucose >250 mg/dL (though this threshold is increasingly de-emphasized due to euglycemic DKA), venous pH <7.3, and serum bicarbonate <15 mEq/L, with elevated blood β-hydroxybutyrate. 1, 2

Core Diagnostic Criteria

All three components must be present simultaneously for DKA diagnosis:

  • Hyperglycemia: Blood glucose >250 mg/dL, though this threshold has been de-emphasized in recent guidelines due to increasing incidence of euglycemic DKA, particularly in patients on SGLT2 inhibitors 1, 3
  • Metabolic acidosis: Venous pH <7.3 AND serum bicarbonate <15 mEq/L 1, 2
  • Ketosis: Elevated blood β-hydroxybutyrate (β-OHB), which is the preferred ketone measurement—NOT urine ketones or nitroprusside-based tests 1, 4
  • Anion gap: Should be >10-12 mEq/L, calculated as [Na⁺] - ([Cl⁻] + [HCO₃⁻]) 1, 2

Severity Classification

DKA severity is classified into three categories based on pH, bicarbonate, anion gap, and mental status, which directly correlates with ICU admission rates, mortality, and healthcare costs. 1, 5

Mild DKA

  • Venous pH: 7.25-7.30 1, 2
  • Serum bicarbonate: 15-18 mEq/L 1, 2
  • Anion gap: >10 mEq/L 1
  • Mental status: Alert 1, 2

Moderate DKA

  • Venous pH: 7.00-7.24 1, 2
  • Serum bicarbonate: 10-15 mEq/L 1, 2
  • Anion gap: >12 mEq/L 1
  • Mental status: Drowsy/lethargic 1, 2
  • ICU care required in 6.7% of cases 5
  • Mortality rate: 13.3% 5

Severe DKA

  • Venous pH: <7.00 1, 2
  • Serum bicarbonate: <10 mEq/L 1, 2
  • Anion gap: >12 mEq/L 1
  • Mental status: Stuporous or comatose 1, 2
  • ICU care required in 47.4% of cases 5
  • Invasive ventilation required in 47% of cases 5
  • Mortality rate: 26% 5
  • Mean healthcare expenditure significantly higher (₹64,000 vs ₹29,000-30,000 for mild/moderate) 5

Essential Laboratory Workup for Classification

  • Complete metabolic panel (sodium, potassium, chloride, bicarbonate, BUN, creatinine, glucose) 1
  • Venous blood gas (pH, pCO2, bicarbonate) 1, 2
  • Blood β-hydroxybutyrate measurement (gold standard)—NOT urine ketones 1, 4
  • Complete blood count with differential 1, 4
  • Urinalysis 1, 4
  • Serum osmolality 1, 4
  • Electrocardiogram 1, 4
  • Anion gap calculation 1, 2

Special Classification Considerations

Euglycemic DKA

  • Blood glucose may be <250 mg/dL while still meeting criteria for DKA (pH <7.3, bicarbonate <15 mEq/L, elevated ketones) 1, 3
  • Increasingly common with SGLT2 inhibitor use 1, 4, 6
  • Same severity classification applies based on pH and bicarbonate levels 6

Diabetic Ketoalkalosis

  • A commonly overlooked alkalemic variant where pH >7.4 despite presence of ketoacidosis 7
  • Accounts for 23.3% of DKA presentations 7
  • All cases have increased anion gap metabolic acidosis present 7
  • 34% have severe ketoacidosis (β-OHB ≥3 mmol/L) requiring same treatment as traditional DKA 7
  • Associated with concurrent metabolic alkalosis (47.2%), respiratory alkalosis (81.1%), or respiratory acidosis (11.3%) 7

Critical Pitfalls to Avoid in Classification

  • Never rely on urine ketones or nitroprusside-based tests for diagnosis or classification—these only measure acetoacetate and acetone, completely missing β-hydroxybutyrate, the predominant ketone in DKA 1, 4
  • Do not dismiss DKA because glucose is <250 mg/dL—euglycemic DKA is increasingly common, especially with SGLT2 inhibitors 1, 4
  • Do not overlook diabetic ketoalkalosis—patients with pH >7.4 can still have severe ketoacidosis requiring full DKA treatment 7
  • Nitroprusside tests can paradoxically worsen during treatment as β-hydroxybutyrate converts to acetoacetate, falsely suggesting worsening ketosis 1, 4

Resolution Criteria (When DKA Classification No Longer Applies)

DKA is resolved when ALL of the following are met:

  • Glucose <200 mg/dL 1, 2
  • Venous pH >7.3 1, 2
  • Serum bicarbonate ≥18 mEq/L 1, 2
  • Anion gap ≤12 mEq/L 1, 2
  • Ketonemia typically takes longer to clear than hyperglycemia, requiring continued monitoring even after glucose normalizes 1, 2

References

Guideline

Diagnosing Diabetic Ketoacidosis (DKA)

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Diagnostic Criteria and Management of Diabetic Ketoacidosis (DKA)

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Research

Diabetic Ketoacidosis: Evaluation and Treatment.

American family physician, 2024

Guideline

Diagnosing Diabetic Ketoacidosis (DKA) in Type 2 Diabetes

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Euglycemic diabetic ketoacidosis in the era of SGLT-2 inhibitors.

BMJ open diabetes research & care, 2023

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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